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Otago HSFY chat - archive

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Pretty much- in NZ we have none of this "entry from high school" rubbish- instead provided you get into university, it's a clean slate for one hard out year of competition.

idk, I just like the sound of it better than putting all that weight on high school
 
ohh... is that the "year 13" thing. sounds cool... doing premed
 
ohh... is that the "year 13" thing. sounds cool... doing premed

No. In NZ, we name our highschool years differently to Aus. Year 13 is the last year of high school in NZ. The first year of uni is either the Biomed/Healthsci/HSFY year depending on which course and which uni you are at.
 
ohh... is that the "year 13" thing. sounds cool... doing premed

NZ year 13 is the same as Aus year 12. The way undergraduate entry for med works in NZ though is basically that the first year of the medical degree is open entry (at Otago this year is called HSFY) but there is a MASSIVE cut in numbers allowed to do second year (and entry to second year is based only on performance in first year - performance in school doesn't matter).
 
hey in the 2008 hubs paper Q15) to contain the infection is this dendritic cells carrying antigen in lymp to lymph nodes where they present leading to activation of CD4+ --> B cells etc
or stages of inflammatory ? i.e. cytokines released from macrophages, mast cells releasing pro-inflammatory mediators and resulting phagocytes comming into the area and destroying the microbes?

im a little confused on which the situation refers to... cheers for any help
 
hey in the 2008 hubs paper Q15) to contain the infection is this dendritic cells carrying antigen in lymp to lymph nodes where they present leading to activation of CD4+ --> B cells etc
or stages of inflammatory ? i.e. cytokines released from macrophages, mast cells releasing pro-inflammatory mediators and resulting phagocytes comming into the area and destroying the microbes?

im a little confused on which the situation refers to... cheers for any help

Think about the timeframe for each of the two processes you have described, and what the bacteria could do in the time it would take for each to work.
 
ah ok right.. so adaptive is slower and obviously innate is first line of defense so to contain the infection = phagocytosis right?

so would this "process" be just phagocytosis or would i need to write about cytokine release from macrophages and anti-imflammatory mediators resulting in chemotaxis which brings the actual phagocytes to the site of infection and THEN explain the process of phagocytosis?
 
O.O haha good to see everyone is going hard for the first semester exam! I know this is a med forum but still....does anyone know how the interviewer score you on dentistry interview questions. Roughly how many questions did they ask and what is the minimum umat needed last year to score interview. Thnx in advance for all the help (yy)
 
hey does somone mind helping me out with understanding the previous exam questions in terms of what they want, ive spent hours revising my notes and i know the content(apparently....) but im finding it so frustrating to interpret the Immunology section of the exam..

like the question i posted before?

cheers
 
I know most healthscis will be cramming like crazy right now, but how'd HUBS go guys?

Was alright....

MCQ- most were easy, some were tricky. I think i just absolutely did not know the answer for like 3 out of the 60. :S

SAQ- It was like the past year papers, except we had a gait cycle question and it started off with immunology question rather identifying the fractured bone like i've seen in the past year papers

Mini Essays- One on cortisol secretion , one on phagocytosis, one on action potential and one on demyelination.
 
Greenglacier had to knock us down when we were talking about questions after the exam! - he reckoned that cruciate ligaments are extracapsular, not intracapsular. My notes said yes, his said no...

But overall a good test.
 
Greenglacier had to knock us down when we were talking about questions after the exam! - he reckoned that cruciate ligaments are extracapsular, not intracapsular. My notes said yes, his said no...

But overall a good test.


Lol, i thought it was intracapsular :S
 
Posteriorly, the femoral attachment of the synovial membrane is located at the cartilaginous margin of the lateral and medial femoral condyles, why the joint space has two dorsal extensions. Between these, the synovial membrane passes in front of the anterior and posterior cruciate ligaments, why these ligaments are both intracapsular and extra-articular with their tibial attachment located exactly on the cartilage margin. Both the lateral and medial meniscus are, however, located within the synovial capsule.

But on another site in wiki they said that both are intracapsular! lol
 
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The HUBS lecture slides call the cruciate ligaments extracapsular and the collateral ligaments capsular.

I was told that the question asked which intracapsular ligament of the knee would be most likely to be damaged by posterior displacement of the fibula, in which case I would say lateral collateral.
 
The HUBS lecture slides call the cruciate ligaments extracapsular and the collateral ligaments capsular.

I was told that the question asked which intracapsular ligament of the knee would be most likely to be damaged by posterior displacement of the fibula, in which case I would say lateral collateral.

I'm pretty sure it was tibia, but either way it's a one mark question.

This year's slides said intracapsular. Let's not worry about it anyway!
 
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